Metabolic alterations related to T2D are well characterised by epidemiological and research centered reports. The pathogenesis and development of T2D is ascribed to four mechanisms; elevated Superior glycation end products (AGE) formation, increased polyol pathway flux, activation of protein kinase C (PKC) isoforms, and amplified hexosamine pathway flux[seven]. Till not https://www.directivepublications.org/journal-of-diabetology-research/